Sample / ModelEvidence (Microglia activation / Inflammasome / Downstream (cytokine) / Cell death / DA (& metabolites) / aSyn)
mouse primary microglia (Gordon, 2018 #586) [mouse primary microglia] MCC950 → i) ASC (이미) monomeric ASC induced by aSyn, fig3c,d), (Oligomeric ASC induced by aSyn, fig3ef), induced by ATP (FIG s6f), induced by nigericin (fig s6f) ii) ↓ cleaved caspase-1 (fig. S6A figs6d, fig3c,d),

(Trudler, 2021 #1900) i) fig 2e, : siNLRP3 (KD)→ ↓ (oligomeric aSyn) -induced IL-1b
i) fig s3, : MCC950 → ↓ (oligomeric aSyn) -induced IL-1b

(Gordon, 2018 #586) [mouse primary microglia] IL-1b (induced by ATP, fig3a) (Induced by fibrillar aSyn, fig3b), induced by ATP (FIG s6e), induced by nig (fig s6e)

(inhouse) TR06673219 → ↓ IL-1b
human iPSC-derived microglia (from normal fibroblast)
(Control) hiPSC-derived microglia/neurons co-culture
Casp1 inhibition (Wang, 2016 #2086)(Wang, 2016 #2086)
Mice / aSyn pff (Gordon, 2018 #586) MCC950 → ↓ cleaved caspase-1 in the striatum of α-syn PFF-injected mice at 30 days (80% correction 30일간 투여)(Fig. 3H).

(Gordon, 2018 #586) aSyn pff, MCC950 (po, 0.3 mg/ml, 8m) → fig6, ↓ (100% correction) NLRP3 (SN, WB), ↓ (90% correction) ASC (SN, WB),

(Gordon, 2018 #586) (fig5) aSyn PFF model : counts of dopaminergic neurons in SN 6m 투여→ 반 correction

(Gordon, 2018 #586) PFF:MCC950 → striatal dopamine concentrations (P < 0.01; Fig. 5H),
NLRP3 KO
AAV-A53T-aSyn model, (prophylactic) (AAV1/2) → 1d 후 MCC950 (10w간) → kill / Uni Wurzburg the NLRP3 activation and TH loss were observed. MCC950 showed 50% reduction of NLRP3 activation after 20 mg/kg ip injection for 10 weeks

MCC950 → (~100%) neuroprotection after 20 mg/kg ip injection for 10 weeks

Inline figure (kept as evidence in `body_r02_c01.jpg`): bar chart `NEW! NLRP3 cells in SN` with annotations `3-fold` and `~50%` reduction; second bar chart panel `Validation of role of NLRP3 inhibition in neuroinflammation` (kept as evidence in `body_r02_c02.jpg`).
AAV-A53T-aSyn mouse(Karikari 2022) IL-2 이 surgery 이후 5주께 증가이긴 하나, 기타 inflammation makrer 들 negative or 불명
α-SYN PFF mouse[Astellas] (symptomatic) model이네 α-SYN PFF dosed into the left striatum (for 13-14 wk → NLRP3 inh 4w) → Hanging wire test
ATG5 KO mice (Cheng, 2020 #1645) MCC950 (10mpk, 1m) → ↓ (60% correction) IBA1+ cells (SN)(fig5f)

(Cheng, 2020 #1645) MCC950 (10mpk, 1m) → ↓ (60% correction) cleaved caspase-1, (fig5c)

(Cheng, 2020 #1645) MCC950 → ↓ (100% correction) MIF (WB, fig5D), ↓ (100% correction) IL-1b mRNA,

(Cheng, 2020 #1645) MCC950 → ↑ (90% correction) TH+Neurons (sn) (fig5j)
aSyn PFF / Nlrp3 inhibitor 아님. (Panicker, 2022 #2067) casp1 KO → ↑TH neurons (fig6h, ~half)

(Panicker, 2022 #2067) casp1 KO → ↑TH (WB)(fig6k, ~half)
Bz-ATPInhouse, BzATP-treated mice : Inzomelid & TR06673219 → ↓ IL-1b
6OHDA (Gordon, 2018 #586) MCC950 20 mpk → 6-OHDA model of PD (80% correction of p20 casp1, 투여기간 7day)(Fig. 3J).

(Gordon, 2018 #586) 6OHDA Mice: DA neurons in SN (Fig. 4, H and I), MCC950 4w 투여→ 반 correction

(Gordon, 2018 #586) MCC950 → ↓ striatal DA & DOPAC, HVA (이것들 다 100% correction, fig. 4, C to E) striatal (TH...
Mitopark(Gordon, 2018 #586) MCC950 20 mpk → in MitoPark mice (투여기간 불상)(Fig. 3I)
Our plan; Atuka / (Intanigral) AAV-A53T-aSyn model,TR0993, TR098, and TR660, (3-doses for) 2.5 months
NLRP3 KO+MPTP mice(Ou, 2021 #1740) NLRP3 KO → protection of:::::: ↓ PD pathology (TH+ cells in SN (70% correction), DA ((90% correction), HVA ((60% correction), caspase-1 ((90% correction), NLRP3 (90% correction), IB-1b (90% correction), TNF-a, IL-...
NLRP3 KO+aSyn mice이게 부재!

Cf) BzATP: 2’(3’)-O-(4-benzoylbenzoyl) adenosine 5’-triphosphate (, P2X7 agonist), {Bae, 2017 #1285} BzATP→ ↑ NLRP3 expression/activation, pro-IL-1β Active-form IL-1β (17kDa), caspase-1 p20

Pathogen 청소 모델Downstream비고
Pathogen이 없어져가는 모델, : bzATP (ivc에 최적은 아니구나)동료모집기능은 전천히 막는건지 모르겠으나,Downatreatm 은 쌓인 것을 말끔히 치움.
P1b: aSyn 불면 모델
P1b의 관건은 기왕것 청소 능력! 이에 최유사 동물모델은?
aSyn 계속 있어, NLRP3 level도 계속 있는 상태BzATP model 에서 ㅂ였듯이, 약이 NLRP3의 하류기능을 빨리 막고, (가정) 동료모집기능은 전천히 막는건가?
P2: TH사망은 비가역적이므로, i) 기왕청소를 한다면, Halting 까지가 가능할 것, reversal 은 불능, ii) 기왕청소 못 하면, slowing 이 목표.
예방 모델: WurzburaSyn 증가중
예방모델: ATG5 (conditional) KO불량 lysosome 출현했지만,약이 nlrp3의 동료모집 기능을 막음 (반). → nlrp3 level 이 반만 증가
예방vs치료 불명Pathogen이 매우 천천히 없어져가는 모델, : 6ohda동료모집기능은 전천히 막는건지 모르겠으나,Downatreatm 은 쌓인 것을 7일만에 (거의) 말끔히 치움. (Gordon, 2018 #586) fig 3J

Uncertain Spans

locationtranscriptionuncertainty
central matrix / column placementthe central matrix has six target columns (Microglia activation / Inflammasome / Downstream (cytokine) / Cell death / DA (& metabolites) / aSyn) per the status-bar body strip; in the main body the cell boundaries are partly cut off and the per-target citation column placement is collapsed into one logical Evidence cell per model row.column placement marked uncertain; original separation should be re-derived by reading the same table on adjacent photos.
Mice / aSyn pff cellreads (Gordon, 2018 #586) MCC950 → ↓ cleaved caspase-1 in the striatum of α-syn PFF-injected mice at 30 days (80% correction 30일간 투여)(Fig. 3H). and the next paragraph reads (po, 0.3 mg/ml, 8m); the 8m token is highlighted yellow.low confidence on whether 8m denotes 8 months or 8 weeks.
Astellas rowreads [Astellas] (symptomatic) model이네 α-SYN PFF dosed into the left striatum (for 13-14 wk → NLRP3 inh 4w) → Hanging wire test; the 13-14 wk token is partly clipped.low confidence on 13-14 wk interval.
bottom Korean table / 약 column entriesthe leftmost column entries , are visible single Korean characters that may denote drug-state labels (medium / severe?); preserved verbatim.low confidence on intended meaning.